関西医大から京都府立医大への道 【7】

 

Retraction Watch / 2011.07.03 00:40 / 推薦数 : 4

今回でシリーズ7回目。

6回目までは、ずっと関西医大発の論文でしたが、今回ようやく京都府立医大にたどりつきました。

先発論文は2003年関西医大のグループより、

後発論文は2005年京都府立医大のグループよりの発表です。

 

京府医大　Previous investigations have provided inferential evidence that biological processes modulated by NO might extend to include angiogenesis.

関西医大　Previous investigations have provided inferential evidence that biological processes modulated by NO might extend to include angiogenesis.

京府医大　We have also reported that overexpression of endothelial NO synthase (eNOS) causes a marked increase in neocapillary formation in response to tissue ischemia.

関西医大　Our study demonstrates that overexpression of eNOS protein causes a marked increase in neocapillary formation in response to tissue ischemia without affecting ischemia-induced VEGF expression or VEGF-mediated Akt phosphorylation.

京府医大　Previous studies reported that VEGF stimulates the release of NO from the arterial wall and promotes the recovery of disturbed endothelium-dependent flow in the ischemic hindlimb.

関西医大　In vitro studies demonstrated that VEGF stimulates the release of NO from the normal arterial wall and promotes the recovery of disturbed endothelium-dependent flow in the rabbit ischemic hindlimb.

京府医大　Direct in vitro evidence that NO may induce angiogenesis was demonstrated recently by Papapetropoulos et al.  Ziche et al established the first line of evidence that NO can induce angiogenesis in vitro.  Murohara et al clearly showedNO-mediated angiogenesis in response to tissue ischemia in NO-deficient mice.

関西医大　Direct in vitro evidence that NO may induce angiogenesis was demonstrated recently by Papapetropoulos et al.  Ziche et al established the first line of evidence that NO can induce angiogenesis in vitro.  Murohara et al clearly showed NO-mediated angiogenesis in response to tissue ischemia by using NO-deficient mice.

 

両論文とも責任著者は同一人物のようです。

次回でこのシリーズ最終回となります。

超一流雑誌の論文が登場します。